I been doing some work on getting a better understanding of peroneal muscle inhibition.
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I see it clinically; I see some dramatic increases in muscle strength testing with the proximal and distal tib-fib mobilization.
I see the symptoms improve.
HOWEVER, I struggle with the understanding of the mechanisms involved and without a coherent explanation, the whole concept runs the risk of being out into the "magical thinking" category by scientists.
I acknowledge that the lack of objective data on the concept is a real issue.
Anyone want to offer up a mechanism or causal pathway between the restrictions at the tib-fib joints and an inhibition of the peroneal longus muscle to work?
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