Diffuse Idiopathic Skeletal Hyperostosis

timharmey · Aug 22, 2011

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I have seen a patient with the above just thought I would flag It up for interest was a new one to me any one who has treated such a patient if you would like to share that would be good.They where referred in with plantar fasciitis
Tim

Craig Payne · Aug 23, 2011

Difficult one ... DISH is associated with really big plantar heel spurs and you often see it written that that is a problem. Most I seen with DISH did not have heel pain and some even had a palpable spur present.

Plantar fasciitis is common in the general population, so always keep in mind that if a DISH patient has heel pain, they could just be getting the pain from a chance occurrence of plantar fasiitis and nothing to do with the DISH. .... or it could be the body spur ...

timharmey · Aug 23, 2011

Thanks , thats useful , I was not sure if the patient having DISH would make the presence of a spur any more significant than usual .
Tim

nigelroberts · Aug 26, 2011

Hi Tim
Having spent many years working with a Rheumatology team, I would agree with Craig and treat the patient as you would if the patient did not have DISH.
Regards
Nigel

admin · Aug 26, 2011

Diffuse idiopathic skeletal hyperostosis

Medical condition

Diffuse idiopathic skeletal hyperostosis (DISH) is a condition characterized by abnormal calcification/bone formation (hyperostosis) of the soft tissues surrounding the joints of the spine, and also of the peripheral or appendicular skeleton.^[1] In the spine, there is bone formation along the anterior longitudinal ligament and sometimes the posterior longitudinal ligament, which may lead to partial or complete fusion of adjacent vertebrae. The facet and sacroiliac joints tend to be uninvolved. The thoracic spine is the most common level involved.^[2] In the peripheral skeleton, DISH manifests as a calcific enthesopathy, with pathologic bone formation at sites where ligaments and tendons attach to bone.

^ Resnick, D.; Shaul, S. R.; Robins, J. M. (June 1975). "Diffuse idiopathic skeletal hyperostosis (DISH): Forestier's disease with extraspinal manifestations". Radiology. 115 (3): 513–524. doi:10.1148/15.3.513. ISSN 0033-8419. PMID 1129458.

^ Sarwark, John, ed. (2010). "Diffuse Idiopathic Skeletal Hyperostosis". Essentials of Musculoskeletal Care (4 ed.). Rosemont, IL: American Academy of Orthopaedic Surgeons. ISBN 978-0-89203-579-3.

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NewsBot · Dec 3, 2012

Press Release:
Western University researchers make breakthrough in arthritis research
Researchers at Western University have made a breakthrough that could lead to a better understanding of a common form of arthritis that, until now, has eluded scientists.

According to The Arthritis Society, the second most common form of arthritis after osteoarthritis is "diffuse idiopathic skeletal hyperostosis" or DISH. It affects between six and 12 percent of North Americans, usually people older than 50. DISH is classified as a form of degenerative arthritis and is characterized by the formation of excessive mineral deposits along the sides of the vertebrae in the neck and back. Symptoms of DISH include spine pain and stiffness and in advanced cases, difficulty swallowing and damage to spinal nerves. The cause of DISH is unknown and there are no specific treatments.

Now researchers at Western University's Bone and Joint Initiative, with collaborator Doo-Sup Choi at the Mayo Clinic in Rochester, Minnesota have discovered the first-ever mouse model of this disease. The research is published online in the Journal of Bone and Mineral Research.

"This model will allow us for the first time to uncover the mechanisms underlying DISH and related disorders. Knowledge of these mechanisms will ultimately allow us to test novel pharmacological treatments to reverse or slow the development of DISH in humans," says corresponding author Cheryle Séguin of the Skeletal Biology Laboratories and the Department of Physiology and Pharmacology at Western's Schulich School of Medicine & Dentistry.

Graduate student Derek Bone, working under the supervision of pharmacologist James Hammond, was studying mice that had been genetically modified to lack a specific membrane protein that transports adenosine when he noticed that these mice developed abnormal calcification (mineralization) of spinal structures.

Changes in the backbone of these mice were characterized by an interdisciplinary team which included: Sumeeta Warraich, Diana Quinonez, Hisataka Ii, Maria Drangova, David Holdsworth and Jeff Dixon. Their findings established that spinal mineralization in these mice resembles DISH in humans and point to a role for adenosine in causing abnormal mineralization in DISH.
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NewsBot · Feb 28, 2017

Talonavicular and Naviculocuneiform Joint Involvement in Diffuse Idiopathic Skeletal Hyperostosis
Misra, Durga Prasanna DM; Chengappa, K. G. MD; Jain, Vikramraj K. DM; Negi, Vir Singh DM
JCR: Journal of Clinical Rheumatology:
March 2017 - Volume 23 - Issue 2 - p 119

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Diffuse idiopathic skeletal hyperostosis

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